I realize that it is a controversial diagnosis. I believe that there is a definitive disease process going on, even if it is not totally understood yet.
Why else would a horse who is not in work, within weeks have both hind legs swell up and drop? First one leg, than while the horse is rested for the first leg, have the second leg swell and drop?
Obviously it isn't injury related, so that means you have to have some sort of disease process going on. My horse was ultrasounded after the first leg swelled up- there were no tears or holes evident on ultrasound. Doesn't that exclude injury? Usually with injuries you have obvious tears on ultrasound examination. Now the ultrasound did show thickening of the ligament.
I don't see the point in doing extensive diagnositic tests. To me any horse who has dropped pasterns in more than one leg, probably has DSLD. Dropped pasterns, enlarged ankles, and a change in hind end comformation, all suggest DSLD to me.
Although a genetic marker hasn't been identified yet doesn't mean it won't be with further research.
"The genome scan identified five chromosomal regions where statistically significant differences were seen between affected and unaffected sample populations that could be indications of linkage to DSLD. Those chromosomes were: ECA 6, 7, 11, 14, and 26.
Further research needs to be conducted especially on ECA 6 and 11 since possible candidate genes are located in those regions based on the human comparative map.